![Figure 4 from Mutant superoxide dismutase 1 (SOD1), a cause of amyotrophic lateral sclerosis, disrupts the recruitment of SMN, the spinal muscular atrophy protein to nuclear Cajal bodies. | Semantic Scholar Figure 4 from Mutant superoxide dismutase 1 (SOD1), a cause of amyotrophic lateral sclerosis, disrupts the recruitment of SMN, the spinal muscular atrophy protein to nuclear Cajal bodies. | Semantic Scholar](https://d3i71xaburhd42.cloudfront.net/e507a7d0797d220d9c7305f4d62ed33899cef525/7-Figure4-1.png)
Figure 4 from Mutant superoxide dismutase 1 (SOD1), a cause of amyotrophic lateral sclerosis, disrupts the recruitment of SMN, the spinal muscular atrophy protein to nuclear Cajal bodies. | Semantic Scholar
![Molecules | Free Full-Text | A Novel Anti-Inflammatory d-Peptide Inhibits Disease Phenotype Progression in an ALS Mouse Model Molecules | Free Full-Text | A Novel Anti-Inflammatory d-Peptide Inhibits Disease Phenotype Progression in an ALS Mouse Model](https://pub.mdpi-res.com/molecules/molecules-26-01590/article_deploy/html/images/molecules-26-01590-ag.png?1615861404)
Molecules | Free Full-Text | A Novel Anti-Inflammatory d-Peptide Inhibits Disease Phenotype Progression in an ALS Mouse Model
![Tempol improves neuroinflammation and delays motor dysfunction in a mouse model (SOD1G93A) of ALS | Journal of Neuroinflammation | Full Text Tempol improves neuroinflammation and delays motor dysfunction in a mouse model (SOD1G93A) of ALS | Journal of Neuroinflammation | Full Text](https://media.springernature.com/lw685/springer-static/image/art%3A10.1186%2Fs12974-019-1598-x/MediaObjects/12974_2019_1598_Fig1_HTML.png)
Tempol improves neuroinflammation and delays motor dysfunction in a mouse model (SOD1G93A) of ALS | Journal of Neuroinflammation | Full Text
![Frontiers | Circadian Rhythm Dysfunction Accelerates Disease Progression in a Mouse Model With Amyotrophic Lateral Sclerosis Frontiers | Circadian Rhythm Dysfunction Accelerates Disease Progression in a Mouse Model With Amyotrophic Lateral Sclerosis](https://www.frontiersin.org/files/Articles/338078/fneur-09-00218-HTML/image_m/fneur-09-00218-g001.jpg)
Frontiers | Circadian Rhythm Dysfunction Accelerates Disease Progression in a Mouse Model With Amyotrophic Lateral Sclerosis
![Humanising mice to enable modelling of neurodegenerative diseases | UCL Queen Square Institute of Neurology - UCL – University College London Humanising mice to enable modelling of neurodegenerative diseases | UCL Queen Square Institute of Neurology - UCL – University College London](https://www.ucl.ac.uk/ion/sites/ion/files/styles/large_image/public/graph_abstract_revised1.jpg?itok=cWnBiFcY)
Humanising mice to enable modelling of neurodegenerative diseases | UCL Queen Square Institute of Neurology - UCL – University College London
![AAV9-mediated gene delivery of MCT1 to oligodendrocytes does not provide a therapeutic benefit in a mouse model of ALS: Molecular Therapy - Methods & Clinical Development AAV9-mediated gene delivery of MCT1 to oligodendrocytes does not provide a therapeutic benefit in a mouse model of ALS: Molecular Therapy - Methods & Clinical Development](https://www.cell.com/cms/attachment/3b365057-92c6-4519-a160-716214c5694a/fx1_lrg.jpg)
AAV9-mediated gene delivery of MCT1 to oligodendrocytes does not provide a therapeutic benefit in a mouse model of ALS: Molecular Therapy - Methods & Clinical Development
![Boosting the peripheral immune response in the skeletal muscles improved motor function in ALS transgenic mice - ScienceDirect Boosting the peripheral immune response in the skeletal muscles improved motor function in ALS transgenic mice - ScienceDirect](https://ars.els-cdn.com/content/image/1-s2.0-S1525001622002489-fx1.jpg)
Boosting the peripheral immune response in the skeletal muscles improved motor function in ALS transgenic mice - ScienceDirect
![MicroRNA-206 Delays ALS Progression and Promotes Regeneration of Neuromuscular Synapses in Mice | Science MicroRNA-206 Delays ALS Progression and Promotes Regeneration of Neuromuscular Synapses in Mice | Science](https://www.science.org/cms/10.1126/science.1181046/asset/ed7b4565-8ab1-4c16-ab4f-93a37e5bcc7c/assets/graphic/326_1549_f2.jpeg)
MicroRNA-206 Delays ALS Progression and Promotes Regeneration of Neuromuscular Synapses in Mice | Science
![Human iPSC-derived neural progenitor cells secreting GDNF provide protection in rodent models of ALS and retinal degeneration - ScienceDirect Human iPSC-derived neural progenitor cells secreting GDNF provide protection in rodent models of ALS and retinal degeneration - ScienceDirect](https://ars.els-cdn.com/content/image/1-s2.0-S2213671123001042-fx1.jpg)
Human iPSC-derived neural progenitor cells secreting GDNF provide protection in rodent models of ALS and retinal degeneration - ScienceDirect
![Motor neuron disease, TDP-43 pathology, and memory deficits in mice expressing ALS–FTD-linked UBQLN2 mutations | PNAS Motor neuron disease, TDP-43 pathology, and memory deficits in mice expressing ALS–FTD-linked UBQLN2 mutations | PNAS](https://www.pnas.org/cms/10.1073/pnas.1608432113/asset/17de35c0-676e-4cc4-b7d4-bb68cc81f735/assets/graphic/pnas.1608432113fig01.jpeg)
Motor neuron disease, TDP-43 pathology, and memory deficits in mice expressing ALS–FTD-linked UBQLN2 mutations | PNAS
![Opinion: more mouse models and more translation needed for ALS | Molecular Neurodegeneration | Full Text Opinion: more mouse models and more translation needed for ALS | Molecular Neurodegeneration | Full Text](https://media.springernature.com/lw685/springer-static/image/art%3A10.1186%2Fs13024-023-00619-2/MediaObjects/13024_2023_619_Fig1_HTML.png)
Opinion: more mouse models and more translation needed for ALS | Molecular Neurodegeneration | Full Text
![A New AAV10-U7-Mediated Gene Therapy Prolongs Survival and Restores Function in an ALS Mouse Model - ScienceDirect A New AAV10-U7-Mediated Gene Therapy Prolongs Survival and Restores Function in an ALS Mouse Model - ScienceDirect](https://ars.els-cdn.com/content/image/1-s2.0-S1525001617302514-fx1.jpg)
A New AAV10-U7-Mediated Gene Therapy Prolongs Survival and Restores Function in an ALS Mouse Model - ScienceDirect
![Identification of novel neuroprotective molecule effective in ALS mouse models | The University of Tokyo Identification of novel neuroprotective molecule effective in ALS mouse models | The University of Tokyo](https://www.u-tokyo.ac.jp/content/400022202.jpg)
Identification of novel neuroprotective molecule effective in ALS mouse models | The University of Tokyo
![TDP-43 transgenic mice develop spastic paralysis and neuronal inclusions characteristic of ALS and frontotemporal lobar degeneration | PNAS TDP-43 transgenic mice develop spastic paralysis and neuronal inclusions characteristic of ALS and frontotemporal lobar degeneration | PNAS](https://www.pnas.org/cms/10.1073/pnas.0912417107/asset/2e535a42-bccf-434f-a669-2405b4a5abaa/assets/graphic/pnas.0912417107fig01.jpeg)